[Lotus]

Hi Sweets, aw thanks, you're pretty awesome too.

Somatostatin inhibits estradiol, GH and aromatase, forskolin too. So, we need to take matters into our hands inhibiting somatostatin (see information below).

As I'll outline the process in upcoming posts it'll start to make sense as to how the menstrual cycle, breast growth and overall health can all live harmoniously in the same universe. 

As you deeper into zinc more of DHT is inhibited, tread carefully though, meaning go slowly monitoring changes (less aggressive, agitation, less stinky, reduction in body hair, etc). For cis-females the dosage will factor around how much androgens your already battling (e.g. BMI, age, what part of the menstrual cycle your in, etc). 

See, LH (luteinizing hormone) is secreted by the anterior pituitary during the proliferative phase (aka-follicular phase).
Why is this important?. Well, LH stimulates androgens from the theca cells thereby converted by granulosa cells, into estrogens. But really?, where do androgens originate?, glad you asked lol, there synthesized from our daily diet,  aka—cholesterol. I'm getting ahead of myself lol because I'll start explaining mitosis and meiosis…however, mitosis is occuring in the granulosa cells during the follicular phase.
Aromatase activity in human granulosa cells during follicular development and the modulation by follicle-stimulating hormone and insulin
https://pubmed.ncbi.nlm.nih.gov/6422764/

In other words Sweets, we don't need more estrogen in the follicular phase...just more conversion of androgens to aromatase. I got the aromatase angle covered though, it'll come via a specific action (or synthesis) to get the job done. 

I wouldn't worry about the loss of DHT and muscle growth. It's more important how you address muscle recovery after working out. I believe a protein synthesis supplement Leucine helps support muscle recovery.

The following information comes by way of the fellas forum regarding inhibiting somatostatin.

On inhibiting somatostatin, it's in breast growers' interest to inhibit its action. Meaning when somatostatin is released it inhibits prolactin, oral estradiol, adenylyl cyclase, growth hormone and TSH (thyroid releasing hormone)...which also inhibits TRH (Thyrotropin releasing hormone). And inhibiting TRH means no prolactin production. In other words prolactin stimulates the aveolargenesis in the lobules of breasts. Vitamin D3 and MSM are very important for alveolar stimulation. 

When you inhibit adenylyl cyclase you inhibit ATP production, and the end result of that is inhibiting aromatase. Insulin inhibits aromatase too. 

See, estrogen stimulates prolactin and growth hormone, so by somatostatin inhibiting their action it seems like an awful waste for breast growth. 

https://www.sciencedirect.com/topics/neu...matostatin

Antibody experiments (1) have clearly shown that basal GH levels are elevated when somatostatin is neutralized. 

Somatomedin C has also been implicated in the negative feedback regulation of GH, at least partially by stimulating release of somatostatin (5). 
https://link.springer.com/chapter/10.100...-7886-4_18

Most effective way to block somatostatin is in the stomach. The release of somatostatin occurs in two places, the pituitary and in the stomach. I believe our best course of action to inhibit somatostatin is in the stomach via the previous post. 

When you inhibit adenylyl cyclase you inhibit ATP production, and the end result of that is it inhibits aromatase. 

Somatostatin is used for treating Gigantism and Acromegaly, and other various conditions:
https://www.drugs.com/drug-class/somatos...alogs.html

Somatostatin agonists for treatment of acromegaly
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2697610/

Thanks for being patient Sweets.