[Lotus]

Thanks MissMad. The following post is also shared here:
https://www.breastnexum.com/showthread.php?tid=17436&page=416

Greetings,

I wanted to introduce (and explain) the first of it's kind model for increasing breast growth using cell energy. Cell's (in the human body) have a lipid bilayer which can either let certain (messages/signals) molecules and ions pass through its receptor sites (on the outside of the cell membrane) or block them.

Fat burning hormones want in...to the nucleus that is. But....when cellular inflammation is occuring outside of this lipid bilayer the signals (messages) won't be able to pass.

What this means is those fat burning hormones unable to synthesize in this scenario is highly inefficient....meaning lost potential for cells to complete its goal of synthesizing DNA into new tissue growth...so to speak.

What causes inflammation of cells?, a number of things quite honestly (e.g. stress, diabetes, poor sleep, poor diet, environmental toxins, cancers, thyroid issues, leaky gut, etc etc...you get the picture)

Get to the point Lotus..? okay okay lol, those (that) fat burning hormones I spoke of are is Leptin. And let's say Leptin makes it way in to the mitochondria (barring mitochondrial dysfunction...which is topic for another discussion) Leptin stimulate aromatase, I'll repeat...stimulating Leptin releases the enzyme aromatase...and as we know aromatase stimulates the conversion of androgens into estrogen.

Leptin explained, a Hormone That Regulates Body Weight. Leptin is a hormone that is produced by your body's fat cells ( 3 ). It is often referred to as the "satiety hormone" or the "starvation hormone." Leptin's primary target is in the brain — particularly ...
https://www.healthline.com/nutrition/leptin-101

Leptin directly stimulates aromatase activity in human luteinized granulosa cells.
https://www.ncbi.nlm.nih.gov/m/pubmed/10421796/

So how do we get this done?, by using exogenous ketones. Exogenous ketones double the electron transport chain and uncoupling protiens thereby improving the mitochondrial proteins in brown fat....think thermogenesis and stimulation of ATP. ATP adenosine triphosphate, is known as cellular currency....ENERGY, and all cells use ATP. I use a HMB (short for β-Hydroxy β-methylbutyric acid) which the one use is a free form-beta-acid-hydroxy-methylbutyrate for my exogenous ketone, it induces protein synthesis through the mTOR pathway and appears to be more active than the using calcium bound butyrate brand.


What Are Exogenous Ketone Supplements?
https://www.healthline.com/nutrition/exo...upplements

Excitatory amino acids activate the enzyme aromatase. Now, estrogen can trigger excitatory neurotransmitters like glutamate, which it appears it will calm the dopamine response. And perhaps one benefit of that calming would reduce food craving. This would help let the Ketones do their thing imo, I think taking 1000 to 1500 mg of glutamine could work though.

Not getting  too far  ahead here, but the branching of breast tissue (aka TEBs- terminal end buds) is a combination of integrins, amphiregulin, metalloproteinase and epidermal growth factor receptors (EGFR). The pituitary stimulates IGF-1 (insulin growth favtor) which then stimulates TEB formation and mammary epithelial branching, but you'll need estrogen for this magic to work. And this activation is using the PrlR/Jak2 pathway by utilizing prolactin leads to the induction of other signaling pathways, such as the mitogen-activated protein kinase (MAPK) and PI 3-kinase (PI3K) pathways. And whoever said prolactin has nothing to do with breast growth is (sorry to say) just plain nuts lol.

Sheesh...look at the time, gotta run. But let the revolution begin.