06-05-2016, 21:04
Thanks stevenator. 
FG is still on the table, I'm not excluding it, just when to take it. Also, find a a standerized version of FG (over 50% saponins). Agree with Abi I do, shatavari is versatile, so is FG (both will raise PRL).
Raising prolactin means lowering (inhibiting) dopamine, specifically @ the inhibition of D2 receptors:
Conventional antipsychotic agents and some, but not all, of the marketed novel agents, elevate serum prolactin levels via inhibition of dopamine action at receptors in the tuberoinfundibular system of hypothalamus, where prolactin secretion is regulated. Specifically, the neurotransmitter dopamine, which acts as the primary prolactin inhibiting factor, is provided to the pituitary gland by the dopaminergic neurons of the periventricular and arcuate nuclei of the medial basal hypothalamus, through the pituitary venus system [1, 9, 10]. Dopamine stimulates receptors located on the surface of the lactotroph pituitary cells and provokes a tonic suppression on prolactin secretion. On the other hand, serotonin stimulates prolactin release [5, 9, 11]. In addition, neuropeptides such as thyrotropin releasing hormone TRH, oxytocin, vasoactive intestinal polypeptide VIP, and peptide histidine-methionine, which are under the control of serotonin, promote prolactin (PRL) secretion.
http://www.hindawi.com/journals/schizort/2013/502697/
Fenugreek raises prolactin, so does anti-psychotics, vitamin B5, gluten (yes gluten), soy products.
Vitamin B5
https://www.healthaliciousness.com/articles/foods-high-in-pantothenic-acid-vitamin-B5.php
Gluten exorphin B5 stimulates prolactin secretion through opioid receptors located outside the blood-brain barrier.
Beta blockers are competitive antagonists that block the receptor sites for the endogenous catecholamines epinephrine (adrenaline) and norepinephrine (noradrenaline) on adrenergic beta receptors, of the sympathetic nervous system, which mediates the fight-or-flight response.[4][5] Some block activation of all types of β-adrenergic receptors and others are selective for one of the three known types of beta receptors, designated β1, β2 and β3 receptors.[6] β1-adrenergic receptors are located mainly in the heart and in the kidneys.[5] β2-adrenergic receptors are located mainly in the lungs, gastrointestinal tract, liver, uterus, vascular smooth muscle, and skeletal muscle.[5] β3-adrenergic receptors are located in fat cells.[7]
Sorry, I'll post more asap.
FG is still on the table, I'm not excluding it, just when to take it. Also, find a a standerized version of FG (over 50% saponins). Agree with Abi I do,
shatavari is versatile, so is FG (both will raise PRL).Raising prolactin means lowering (inhibiting) dopamine, specifically @ the inhibition of D2 receptors:
Conventional antipsychotic agents and some, but not all, of the marketed novel agents, elevate serum prolactin levels via inhibition of dopamine action at receptors in the tuberoinfundibular system of hypothalamus, where prolactin secretion is regulated. Specifically, the neurotransmitter dopamine, which acts as the primary prolactin inhibiting factor, is provided to the pituitary gland by the dopaminergic neurons of the periventricular and arcuate nuclei of the medial basal hypothalamus, through the pituitary venus system [1, 9, 10]. Dopamine stimulates receptors located on the surface of the lactotroph pituitary cells and provokes a tonic suppression on prolactin secretion. On the other hand, serotonin stimulates prolactin release [5, 9, 11]. In addition, neuropeptides such as thyrotropin releasing hormone TRH, oxytocin, vasoactive intestinal polypeptide VIP, and peptide histidine-methionine, which are under the control of serotonin, promote prolactin (PRL) secretion.
http://www.hindawi.com/journals/schizort/2013/502697/
Fenugreek raises prolactin, so does anti-psychotics, vitamin B5, gluten (yes gluten), soy products.
Vitamin B5
https://www.healthaliciousness.com/articles/foods-high-in-pantothenic-acid-vitamin-B5.php
Gluten exorphin B5 stimulates prolactin secretion through opioid receptors located outside the blood-brain barrier.
Beta blockers are competitive antagonists that block the receptor sites for the endogenous catecholamines epinephrine (adrenaline) and norepinephrine (noradrenaline) on adrenergic beta receptors, of the sympathetic nervous system, which mediates the fight-or-flight response.[4][5] Some block activation of all types of β-adrenergic receptors and others are selective for one of the three known types of beta receptors, designated β1, β2 and β3 receptors.[6] β1-adrenergic receptors are located mainly in the heart and in the kidneys.[5] β2-adrenergic receptors are located mainly in the lungs, gastrointestinal tract, liver, uterus, vascular smooth muscle, and skeletal muscle.[5] β3-adrenergic receptors are located in fat cells.[7]
Sorry, I'll post more asap.