[Lotus]

If you have specific "Relevant" info concerning what's posted I suggest you provide more research regarding as such rather than personal bias.

I have no bias towards or against either natural or synthetic therapies. I judge each substance on its own merits. I believe that the study I cited and the well-known incurable side-effect of mucuna's active ingredient provide a legitimate reason to warn people away from mucuna.

If I had a prolactin problem that didn't respond to chasteberry, I'd try to get some pramipexole. And I would refuse a prescription for any of the numerous dopamine agonists that are ergot derivatives, because they all carry some risk of heart valve fibrosis.

The study you mention states this:

(Same study, different reporting source)
Medscape Medical News
Mucuna pruriens Seed May Be Helpful in Long-term Management of Parkinson's Disease:

These findings suggest that M pruriens formulations may actually have a higher bioavailability than standard L-dopa preparations.... If these findings can be confirmed in larger and longer term studies, mucuna would seem to be a reasonable commercially viable alternative to standard L-dopa."

http://www.medscape.com/viewarticle/494984

It would make more sense not take herbs that increase Prolactin, like Fenugreek or Goats Rue if your prolactin levels are high. However in any event mucuna pruriens isn't high on my priority list of herbs for NBE.

In fact the study I listed states this:

A number of agents discussed here that have a proposed role in the treatment of neurodegenerative diseases (and PD in particular), including cannabis, mucuna pruriens, and Chinese herbals, deserve more attention from basic science researchers and clinical investigators before they can be either safely utilized or dismissed.

So I'll go by that suggestion.