[hannah]

For all the readers out there, here's some digged up science on peanut oil.
Don't thank me, but Lotus for this.

you said it opened new receptors? Please tell me more, how do we use this nutty combination?:p

Oh that nutty combo? ,  (yes, here it is). First, heads up to peanut allergy suffers, alternatively, replacing with an omega 3 supplement is an option in my opinion, my choice of omega 3 option would be krill oil instead of peanut oil.  

I think peanut oil works because it contains resveratrol (a known aromatase) and nitric oxide (a second messenger, aka promotes DNA synthesis).  EPO (evening primrose oil) inhibits DHT and is a pro-aromatase. Signaling pathways are very interesting, they involve hormones (sex streiods), fatty acids, enzymes (e.g. Acetyl coenzyme A or acetyl-CoA) etc, understanding these pathways helps us to understand how relates to NBE (hrt too)..........and yes it's very complicated, to me it's worth investigating. 

Both linoleate and arachidonate can activate Type-II and Type-III protein kinase-C in MEC

translation—linoleate (i.e. evening primrose oil) and arachidonate (i.e. peanut oil) improves cellular affinity of mammary epithelial.....aka milk duds, I mean milk ducts.  though do add calcium with this combination, it activates the proteins. 

Peanut oil (a DIY home method to make peanut oil is included). 
http://articles.mercola.com/herbal-oils/peanut-oil.aspx

Ok, now we're making some progress, "linoleic acid (18:2 omega 6) and prostaglandin E2 or cAMP has synergistic effect on EGF-stimulated growth". 

(Linoleic acid is evening primrose oil.)

What is epidermal growth factor?
The epidermal growth factor receptor (EGFR; ErbB-1; HER1 in humans) is the cell-surface receptor for members of the epidermal growth factor family (EGF family) of extracellular protein ligands.
Epidermal growth factor receptor - Wikipedia, the free encyclopedia
Wikipedia › wiki › Epidermal_growth_f...


transducers: amplification of signals from growth factor receptors by fatty acids in mammary epithelial cells.

Abstract
The growth, morphogenesis and differentiation of milk producing epithelial tissues in the developing mammary glands require interaction with extracellular matrices and stimulation by hormones, growth factors and essential fatty acids. In primary culture, the proliferation of mammary epithelial cells (MEC), induced by epidermal growth factor (EGF), is enhanced and sustained by linoleate and its eicosanoid metabolites. Since a combination of linoleic acid (18:2 omega 6) and prostaglandin E2 or cAMP has synergistic effect on EGF-stimulated growth, it is suggested that additional cAMP-dependent protein kinase A (PK-A) independent pathways may also contribute to the linoleate effect on EGF action. Possible involvement of Ca2+/phospholipid-dependent protein kinase C (PK-C) is explored. Both linoleate and arachidonate can activate Type-II and Type-III protein kinase-C in MEC and a PK-C inhibitor can block growth stimulation by EGF and fatty acids. Like 12-O-Tetradecanoly phorbol-13-acetate (TPA), a PK-C activator which also enhances EGF-stimulated growth of MEC, linoleate can phosphorylate a 40-42 KD protein. EGF itself can stimulate transient phosphorylation of the same protein in MEC cultures but when supplemented with linoleate, which does not influence the ligand binding affinity of EGF-receptors, the transient phosphorylation signal in 40-42 KD protein is sustained.(ABSTRACT TRUNCATED AT 250 WORDS)

http://www.ncbi.nlm.nih.gov/pubmed/8424125

So its a bit complicated, the useful tip here is.. correct me if I am wrong..combine Epo with peanut oil